Li Lab's Research
Currently, Dr. Li leads a large-scale genomic study funded by the NIDDK, investigating the role of mitochondrial dysfunction in CKD progression among more than 5,000 CKD patients from the Chronic Renal Insufficiency Cohort (CRIC). This study also seeks to identify factors that influence mitochondrial function in CKD patients. Additionally, he is spearheading a metabolomics study of 1,500 CKD patients of African ancestry to uncover factors that trigger the adverse effects of APOL1 gene mutations on CKD progression.
Currently, Dr. Li leads a large-scale genomic study funded by the NIDDK, investigating the role of mitochondrial dysfunction in CKD progression among more than 5,000 CKD patients from the Chronic Renal Insufficiency Cohort (CRIC). This study also seeks to identify factors that influence mitochondrial function in CKD patients. Additionally, he is spearheading a metabolomics study of 1,500 CKD patients of African ancestry to uncover factors that trigger the adverse effects of APOL1 gene mutations on CKD progression.
Currently, Dr. Li leads a large-scale genomic study funded by the NIDDK, investigating the role of mitochondrial dysfunction in CKD progression among more than 5,000 CKD patients from the Chronic Renal Insufficiency Cohort (CRIC). This study also seeks to identify factors that influence mitochondrial function in CKD patients. Additionally, he is spearheading a metabolomics study of 1,500 CKD patients of African ancestry to uncover factors that trigger the adverse effects of APOL1 gene mutations on CKD progression.
Currently, Dr. Li leads a large-scale genomic study funded by the NIDDK, investigating the role of mitochondrial dysfunction in CKD progression among more than 5,000 CKD patients from the Chronic Renal Insufficiency Cohort (CRIC). This study also seeks to identify factors that influence mitochondrial function in CKD patients. Additionally, he is spearheading a metabolomics study of 1,500 CKD patients of African ancestry to uncover factors that trigger the adverse effects of APOL1 gene mutations on CKD progression.