The Ding lab is led by Dr. Kan Ding, an Associate Professor and practicing neurologist who specializes in epilepsy. The lab concentrates on neurocritical care and post-traumatic epilepsy, with the goal of advancing strategies for diagnosis, treatment, and rehabilitation to improve care for those with epilepsy. 

The Sguigna lab investigates the visual system in multiple sclerosis, and other neurological conditions, with the intent of leveraging technologies to further science both diagnostically and therapeutically. 

Heart-Brain Connection Program

The Monson Lab is dedicated to understanding how B cells and T cells impact pathology of disease in the central nervous system.

Our mission is to better unravel the causes and mechanisms underlying tremor disorders as well as understand the clinical features of these disorders.

We are interested in the circuit mechanisms of how the cerebellum helps the brain to work better in health and disease.

The Greenberg Lab focuses on translational research relative to autoimmune disorders of the central nervous system.

The vision of the lab is to further understand the pathogenesis of autoimmunity of the central nervous system through basic science and translational research.

The focus of the Neuromuscular Center is the diagnosis and treatment of muscle diseases known as metabolic myopathies, including inherited disorders of muscle fat, carbohydrate, and mitochondrial muscle metabolism.

The central goal of the Dauer Lab is to unravel the molecular and cellular mechanisms of diseases that disrupt the motor system. In exploring these diseases, we also aim to understand a fundamental question relevant to CNS disease generally: what factors determine the selective vulnerability of particular cell types or circuits to insults? Our primary focus is on Parkinson’s disease and inherited forms of dystonia. We focus our efforts on disease genes that cause these disorders, employing a range of molecular, cellular, and whole animal studies to dissect the normal role of disease proteins, and how pathogenic mutations lead to disease.